Barry H. Paw , MD, PhD
hematopoiesis and iron metabolism
Associate Professor of Medicine, Harvard Medical School
Pediatrics, Pediatric Hematology/Oncology
A.B., Biochemistry, University of California, Berkeley, Berkeley, CA (1984)
Ph.D., Biological Chemistry, University of California, Los Angeles School of Med, Los Angeles, CA (1991)
M.D., Medicine, University of California, Los Angeles School of Medicine, Los Angeles, CA (1991)
Pediatrics, Children's Hospital Boston, Boston, MA (1991-1992)
Pediatrics, Boston Combined Residency Program, Children's Hospital Boston/Boston Medical Center, Boston, MA (1992-1993)
Pediatric Hematology-Oncology, Children's Hospital Boston and Dana-Farber Cancer Institute, Boston, MA (1993-1996)
Shaw GC, et al.
Nature Issue: 440 , Page: 96-100
Shafizadeh E & Paw BH
Curr. Opinion Hematol Issue: 11 , Page: 255-261
Paw BH, et al.
Nat. Genet. Issue: 34 , Page: 59-64
Nilsson R, et al..
Cell Metabolism , 2009
Chen W, et al.
Proc. Natl. Acad. Sci. USA , 2009
Chen W, et al.
Blood , in press, 2010
Discovery of genes essential for heme biosynthesis through large-scale gene expression analysis (Cell Metabolism 10:119-30, 2009)
Abcb10 physically interacts with mitoferrin-1 (Slc25a37) to enhance its stability and function in the erythroid mitochondria (Proc Natl Acad Sci U S A. 2009 Sep 22;106(38):16263-8)
Ferrochelatase forms an oligomeric complex with mitoferrin-1 and Abcb10 for erythroid heme biosynthesis (Blood 116:628-30, 2010)